Spring 2017  Saber Tails 75    

Petit Basset Griffon Vendéen Club of America

Cont’d from previous page

benefit of transfusing plasma into dogs with acute pancre-
atitis, unless the dog is exhibiting coagulation problems.
• Anti-emetics are recommended for all dogs with acute 
pancreatitis. Clinical signs of nausea include licking of 
lips, swallowing attempts, and obvious aversion to food. 
Anti-emetics should be given even to those with no overt 
signs of nausea or vomiting because this may encourage 
voluntary eating.
• Pain should be managed. It is safe to assume that all dogs 
with acute pancreatitis are in pain to some degree. In or-
der to manage the pain with the appropriate analgesics, it 
is important to determine the level of pain that the dog is 
experiencing. A dog with mild pain might be responsive 
to his/her surroundings, but be unsettled. With moder-
ate pain, a dog might be reluctant to move and may flinch 
when his/her abdomen is palpated. A dog in severe pain 
may be non-responsive to surroundings and may scream 
or snap when palpated. Most referral veterinary hospitals 
have pain management specialists who can assess the level 
of pain a dog is experiencing and prescribe the appropriate 
combination of analgesic agents.
• Food and water should be offered early in the recovery 
phase. The role of nutrition in treatment of acute pancre-
atitis has changed over the past two decades. It was previ-
ously thought that pancreatitis patients (both human and 
animal) should fast in order to ‘rest’ their pancreas. Cur-
rent recommendation is that dogs with mild acute pancre-
atitis be allowed to eat voluntarily. If the dog does not eat 
for five days, then a nasogastric tube should be inserted 
for enteral feeding of a liquid diet. Dogs with severe acute 
pancreatitis should receive interventional food as soon as 
possible. Generally, during recovery from a bout of pan-
creatitis, a low-fat diet is given for a period of time after 
which a regular diet can be reintroduced (unless the dog 
has high serum triglyceride levels). Once a dog has had 
pancreatitis, the chances of a recurrent event is high. The 
dog’s diet must be monitored vigilantly and his/her treats 
selected with care.

D

ottie was 11 years old when she became agitated, 
started pacing around the room, and began to bite 

the wooden knobs on a dresser. She then stretched her 
body out with her rear in the air. She was taken to the vet 
immediately. Blood tests indicated an acute bout of pan-
creatitis. Ranididine (Zantac) was given to reduce stomach 
acid production and she recovered over the course of a 
week. The cause of her attack was never known, but her 
family kept her on a low-fat diet as a precaution. Whenever 
her mom thought she might be have another attack, she 
was given the Ranididine and it never recurred. She is 14 
now and a hearty eater who loves her walks and a cuddle.

L

essons from the hounds.  What can we learn about 
pancreatitis from our beloved hounds? Several mem-

bers of our collective pack suffered bouts of pancreatitis 
after a high-fat feast, either a cup of chowder or some pup-
py food or a delicious home cooked meal with too much fat. 
For others, there was no known dietary indiscretion, but it 
is reasonable to assume that fat was a contributing trigger 
since switching the dogs to a low-fat food and closely mon-
itoring all foods and treats prevented recurrence. Many af-
fected dogs can have good quality of life if their pancre-
atitis is caught early and treated with special diets and/
or medications for the rest of their lives. For other dogs, 
the inflammatory storm rapidly spread to other organs 
and the most advanced veterinary care was in vain. We 
hope that research into the causes of pancreatitis in dogs 
and development of specific treatments will, in the future, 
increase survival of those suffering severe acute attacks.

Abraham is our elder statesman at 17 years old. His 
first bout of pancreatitis occurred when he was 11. His 
mom noticed that he seemed uncomfortable walking and 
wouldn’t lay on his tummy, which was his usually resting 
position. The emergency vet gave him pain meds and per-
formed tests that revealed signs of pancreatitis. The vet 
put him on a bland, unappetizing canned dog food, which 
Abraham totally rejected. His mom experimented with a 
modified Dodds’ liver-cleaning diet and Abraham’s appe-
tite picked up. To this day, he cannot tolerate any fat in his 
diet. He gets three meals a day of a super low fat kibble 
mixed with white fish, dry curd cottage cheese, and baked 
sweet potato. Sometimes he gets boiled chicken instead of 
fish, canned pumpkin instead of sweet potato, and some 
jasmine rice. Sounds yummy to me! Abraham’s family sur-
mises that his attack was triggered by his habits of root-
ing through the kitchen garbage and eating poop. They 
are vigilant on both accounts and, with his diet controlled, 
his chronic condition has been managed. Abraham has had 
a very active life, with Rally Advanced and Utility Dog 
titles. It is wonderful that he can rest easy in his senior 
years, knowing that most of the delicious cooking in the 
household is for him.

References:

AKC.org health article. December 17, 2015. Pancreatitis in dogs – symp-

toms, causes, and treatment. (http://akc.org/content/health/articles/pan-

creatitis-in-dogs/?utm_source=newsletter&utm_medium=email&utm_

campaign=yourakc-20161226); LaRusch J. and Whitcomb, D.C. 2011. 

Genetics of pancreatitis. Current Opinion in Gastroenterology 27: 467-474.; 

Mansfield C. and Beths T. 2015. Management of acute pancreatitis in dogs: 

a critical appraisal with focus on feeding and analgesia. Journal of Small 

Animal Practice 56: 27-39.; Watson, P. 2015. Canine and feline pancreatitis: 

a challenging and enigmatic disease. Journal of Small Animal Practice 56: 

1-2.; Watson, P. 2015. Pancreatitis in dogs and cats: definitions and patho-

physiology. Journal of Small Animal Practice 56: 3-12.; Xenoulis, P.G. 2015. 

Diagnosis of pancreatitis in dogs and cats. Journal of Small Animal Practice 

56: 13-26.